Osteochondrosis and juvenile spavin in equids

Barbara J. Watrous From the College of Veterinary Medicine (Watrous and Wagner), Oregon State University, Corvallis, OR 97331-1802, and the Department of Veterinary Pathobiology (Hultgren†). The University of Minnesota, St Paul, MN 55108.

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Bruce D. Hultgren From the College of Veterinary Medicine (Watrous and Wagner), Oregon State University, Corvallis, OR 97331-1802, and the Department of Veterinary Pathobiology (Hultgren†). The University of Minnesota, St Paul, MN 55108.

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Pamela C. Wagner From the College of Veterinary Medicine (Watrous and Wagner), Oregon State University, Corvallis, OR 97331-1802, and the Department of Veterinary Pathobiology (Hultgren†). The University of Minnesota, St Paul, MN 55108.

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SUMMARY

Thirty-six of 50 young equids examined at necropsy for gross pathologic and histopathologic evidence of osteochondrosis were determined to have lesions characteristic of this disorder in the distal joints of the tarsus. Abnormalities ranged from retained endochondral cores underlying undisturbed articular cartilage surfaces to clefts, subchondral osseous cyst-like lesions, and cartilage ulceration. Our findings supported the conclusion that osteochondrosis may cause spavin in the juvenile equid.

SUMMARY

Thirty-six of 50 young equids examined at necropsy for gross pathologic and histopathologic evidence of osteochondrosis were determined to have lesions characteristic of this disorder in the distal joints of the tarsus. Abnormalities ranged from retained endochondral cores underlying undisturbed articular cartilage surfaces to clefts, subchondral osseous cyst-like lesions, and cartilage ulceration. Our findings supported the conclusion that osteochondrosis may cause spavin in the juvenile equid.

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