A 13-year-old 7.4-kg (16.3-lb) spayed female Miniature Schnauzer underwent a routine recheck evaluation. The dog had had a transvenous permanent pacemaker implanted 3.5 years earlier because of sick sinus syndrome. Depletion of the generator battery necessitated a generator battery change 10 months prior to the recheck evaluation, which was performed with no complications. The pacemaker mode was programmed for ventricular demand rate-responsive pacing (VVIR) with a base rate of 40 beats/min and a maximum rate of 160 beats/min. Previous medical history included American College of Veterinary Internal Medicine classification1 stage B1 degenerative mitral and tricuspid valve disease, a
A 10-year-old 28.0-kg (61.6-lb) spayed female Golden Retriever was evaluated because of a 2-day history of lethargy and anorexia. The dog also had a history of hypothyroidism and idiopathic epilepsy, for which it was receiving levothyroxine and phenobarbital, respectively. In addition, approximately 2 weeks before the examination, forelimb lameness had developed and resolved spontaneously.
On examination, the dog was lethargic and tachypneic (60 breaths/min; reference range, 16 to 40 breaths/min) and had a rectal temperature of 39.4°C (102.9°F). Cardiac auscultation revealed an irregular heart rhythm with a heart rate of 80 beats/min (reference range, 60 to 140 beats/min) and
A 1-year-old 20.2-kg neutered male mixed-breed dog was presented to the emergency service at the BluePearl Pet Hospital in New Braunfels, Texas, because of acute collapse and lethargy. On presentation, the dog was quiet, dull, and responsive. Cardiac auscultation revealed severe tachycardia (350 beats/min) with a regular rhythm and no murmur. Bronchovesicular sounds were considered normal. Femoral pulses were weak, and frequent pulse deficits were detected. A lead II ECG tracing was obtained (Figure 1).
Initial lead II ECG tracing obtained from a 1-year-old mixed-breed dog following an episode of acute collapse and
An 8-year-old spayed female Japanese Chin was referred for evaluation of an acute onset of ataxia and exacerbation of chronic signs of neck pain. The dog had a history of intermittent neck-guarding episodes since acquisition at 2 years of age. Abnormalities on physical examination included advanced periodontal disease, pigmentary keratitis, and corneal scarring of the left eye. The neck was held in ventroflexion. The dog was reluctant to move the neck in dorsal and lateral directions. Observation of the gait revealed mild general proprioceptive ataxia most notable in the right pelvic limb. Postural reactions were delayed in the right
A 3-year-old 392-kg (862-lb) Rocky Mountain filly with a 5-day history of hypoalbuminemia and facial edema that progressed to ventral edema was referred to the University of Missouri Veterinary Medical Teaching Hospital for further diagnostic testing and evaluation. At the initial referral evaluation, the horse was alert and responsive; heart rate was 44 beats/min, respiratory rate was 40 breaths/min, and rectal temperature was 37.8°C (100.0°F). Mild ataxia in the hind limbs was evident. A CBC and plasma biochemical analysis revealed a stress leukogram and profound panhypoproteinemia (albumin concentration, < 1.5 g/dL [reference range, 3.5 to 4.4 g/dL]; total protein concentration,