Search Results

You are looking at 1 - 2 of 2 items for

  • Author or Editor: Ricky G. Cashmore x
  • Refine by Access: All Content x
Clear All Modify Search

Abstract

CASE DESCRIPTION

A 3-year-old 2.5-kg (5.5-lb) sexually intact male Pomeranian was presented with a 1-day history of non–weight-bearing lameness of the right forelimb.

CLINICAL FINDINGS

Signs of pain were localized to the proximal portion of the right antebrachium. Radiography revealed a minimally displaced fracture of the proximal portion of the radius that had propagated from a well-demarcated, ovoid, osteolytic lesion within the cortex of the caudolateral aspect of the radius. Computed tomographic findings supported the radiographic findings and did not reveal lesions in other evaluated body sites.

TREATMENT AND OUTCOME

At surgery, the lateral aspect of the radial cortex appeared expanded, and tenacious fibrous tissue filled the gap between the fracture fragments. Fibrous tissue was resected and submitted for histologic examination, and the fracture was reduced and stabilized with a bone plate and a positional screw. Histologic examination revealed the presence of viable bone, fibrous tissue, and areas of coagulative necrosis. Imaging and histologic findings were consistent with radioulnar ischemic necrosis (RUIN). The patient ambulated normally at reexamination 12 days after surgery. At reexamination 15 weeks after surgery, the patient continued to ambulate normally, and radiography and CT indicated healing of the fracture and resolution of the RUIN lesion.

CLINICAL RELEVANCE

RUIN should be considered as a differential diagnosis for a dog with forelimb lameness and radiographic focal osteolysis between the proximal and middle thirds of the diaphysis of the radius or ulna. Prognosis for dogs with RUIN may be good with surgical intervention.

Full access
in Journal of the American Veterinary Medical Association

Abstract

CASE DESCRIPTION

3 adult Basset Hounds were referred for evaluation of chronic, unilateral, pelvic limb lameness with no history of trauma.

CLINICAL FINDINGS

On examination, all dogs had mild lameness of the affected limb; signs of pain were evident during manipulation of the stifle joint in the affected limb, along with effusion of that joint. No stifle joint instability was palpable. Radiographs were available for review for 2 of the 3 dogs. Effusion was confirmed radiographically, but severity of degenerative joint disease varied. Central intercondylar notch width ratios for the 2 dogs were 0.16 and 0.17, and tibial plateau angles were −10° and 15°; relative tibial tuberosity width was 1.1 for both dogs. Exploratory arthroscopy revealed moderate degeneration of the caudal cruciate ligament in all 3 dogs; the cranial cruciate ligaments were grossly normal.

TREATMENT AND OUTCOME

Corrective osteotomy to increase the tibial plateau angle was performed in 1 dog, and the lameness resolved by 2 months after surgery. The 2 other dogs were managed without additional surgery. One dog was persistently lame. The other dog reportedly had normal limb function 2.5 years after undergoing exploratory arthroscopy.

CLINICAL RELEVANCE

Morphological characteristics of the tibia in Basset Hounds may predispose to abnormal stresses on the caudal cruciate ligament. Isolated degeneration of the caudal cruciate ligament should be considered as a differential diagnosis for Basset Hounds with lameness originating from the stifle joint. Without direct inspection of the joint, caudal cruciate ligament disease could be confused for cranial cruciate ligament injury.

Full access
in Journal of the American Veterinary Medical Association