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- Author or Editor: Britton K. Nixon x
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A 4-year-old 7.3-kg (16-lb) castrated male Pug was evaluated because of a sudden onset of rapidly progressive neurologic abnormalities. Four days prior to referral evaluation, the owner perceived the dog to have right forelimb lameness. The following day, a neurologic examination by the primary care veterinarian revealed ambulatory tetraparesis with decreased conscious proprioception in all limbs. Clinical signs were refractory to empirical fluid therapy administered SC and an NSAID administered orally.
At the referral evaluation, the dog had markedly diminished mentation. Physical examination revealed nonambulatory tetraparesis, vertical nystagmus of the right eye, rotary nystagmus of
