Search Results

You are looking at 1 - 3 of 3 items for

  • Author or Editor: Babetta A. Breuhaus x
  • Refine by Access: All Content x
Clear All Modify Search

Abstract

Objective—To determine whether ingestion of fescue seed infected with the endophyte Neotyphodium coenophialum would alter thyroid function in adult horses.

Design—Original study.

Animals—4 adult mares that were not pregnant and 6 adult geldings.

Procedure—Thyrotropin releasing hormone stimulation tests were performed while horses received a standard diet and after infected seed (2.3 kg/d [5 lb/d]) had been fed for 1 and 2 months. Serum prolactin concentrations were measured to verify endophyte absorption.

Results—Serum prolactin concentrations indicated that at least 8 of 10 horses absorbed the endophyte. Baseline concentrations of thyroid stimulating hormone, total and free triiodothyronine, and total and free thyroxine and the change in hormone concentrations in response to administration of thyrotropin releasing hormone (1 mg, IV) were not altered by ingestion of endophyte-infected fescue seed.

Conclusions and Clinical Relevance—Results suggest that ingestion of fescue seed infected with the endophytic fungus N coenophialum for 2 months has little effect on thyroid function in adult horses that are not pregnant. (J Am Vet Med Assoc 2003;223:340–345)

Restricted access
in Journal of the American Veterinary Medical Association

Abstract

Objective—To identify clinical signs, underlying cardiac conditions, echocardiographic findings, and prognosis for horses with congestive heart failure.

Design—Retrospective study.

Animals—14 horses.

Procedure—Signalment; history; clinical signs; clinicopathologic, echocardiographic, and radiographic findings; treatment; and outcome were determined by reviewing medical records.

Results—All 14 horses were examined because of a heart murmur; tachycardia was identified in all 14. Twelve horses had echocardiographic evidence of enlargement of 1 or more chambers of the heart. Other common clinical findings included jugular distention or pulsation, crackles, cough, tachypnea, and ventral edema. Nine horses had signs consistent with heart failure for > 6 days. Underlying causes for heart failure included congenital defects, traumatic vascular rupture, pericarditis, pulmonary hypertension secondary to heaves, and valvular dysplasia. Seven horses were euthanatized after diagnosis of heart failure; 5 were discharged but were euthanatized or died of complications of heart disease within 1 year after discharge. The remaining 2 horses were discharged but lost to follow-up.

Conclusions and Clinical Relevance—Results suggest that congestive heart failure is rare in horses. A loud heart murmur accompanied by either jugular distention or pulsation, tachycardia, respiratory abnormalities (crackles, cough, tachypnea), and ventral edema were the most common clinical signs. Echocardiography was useful in determining the underlying cause in affected horses. The long-term prognosis for horses with congestive heart failure was grave. (J Am Vet Med Assoc 2002;220:1512–1515)

Restricted access
in Journal of the American Veterinary Medical Association
in Journal of the American Veterinary Medical Association