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proinflammatory cytokines such as interleukin-1β (IL-1β), tumor necrosis factor-α (TNFα), and interferon-γ (IFNγ), 9 as well as increased parathyroid hormone (PTH) levels. 10 A recent study 11 in a rodent CKD model with nonregenerative anemia showed the

Open access
in American Journal of Veterinary Research

closely regulated along with phosphorus via parathyroid hormone (PTH), calcitriol (1,25-dihydroxyvitamin D3), and calcitonin. 1 – 3 During periods of hypocalcemia, PTH secretion increases plasma calcium via mobilization of calcium and phosphorus from bone

Open access
in Journal of the American Veterinary Medical Association

phosphatase activity (230 U/L; age-adjusted reference range, 2 37 to 333 U/L). Urine specific gravity was concentrated at > 1.050. A serum parathyroid hormone (PTH), calcium, and vitamin D profile revealed a mildly high concentration of PTH (33 pg/mL; age

Open access
in Journal of the American Veterinary Medical Association

increase in PTH (parathyroid hormone), which promotes renal and salivary phosphorus excretion as well as mobilization of phosphorus from bone. Hypophosphatemia should therefore self-correct following the treatment of hypocalcemia. 8 The relationship

Open access
in Journal of the American Veterinary Medical Association

osteoclast perspective . Animals . 2022 ; 12 ( 13 ): 1722 . doi: 10.3390/ani12131722 12. Delmas PD , Vergnaud P , Arlot ME , Pastoureau P , Meunier PJ , Nilssen MHL . The anabolic effect of human PTH (1–34) on bone formation is

Open access
in American Journal of Veterinary Research