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expression in the atria, compared with findings for the ventricles. In CDDs, expression of IL-2, IFN-γ, IL-1, and TGF-β1 differed among cardiac regions. Interleukin-2 and IFN-γ mRNA expression was significantly ( P = 0.008 and P = 0.042, respectively

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in American Journal of Veterinary Research

defense mechanism against cellular damage, yet chronic stimulation results in disease progression caused by alternations in energy production, membrane damage, and apoptosis. 10,11 Interleukin-6 and the receptor for IL-1 were upregulated in our study and

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in American Journal of Veterinary Research

than PGF 2α at human FPRs, 12 and that FPRs and EP 1 Rs mediate PGF 2α - and PGE 2 -induced expression of interleukin-1B in a progenitor testicular cell line. 13 Furthermore, PGF 2α binds to EPRs in pigs, 14 both PGF 2α and PGE 2 constrict

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in American Journal of Veterinary Research

that, in an vivo situation, the effect of LPS at the vascular level could occur mainly indirectly through cytokine mediators (tumor necrosis factor and interleukin-1) released from activated leukocytes. In the present study, treatment with LPS reduced

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in American Journal of Veterinary Research

circulating concentrations of prosta-glandins as well as inflammatory cytokines (especially interleukins 1 and 6) can be associated with an increase in plasma ANP concentration. Thus, the relatively high Nt-proANP concentrations in the control cats may be a

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in American Journal of Veterinary Research

. Lee YJ Shin KC Kang SW et al Type III procollagen N-terminal propeptide, soluble interleukin-2 receptor, and von Willebrand factor in systemic sclerosis . Clin Exp Rheumatol 2001 ; 19 : 69 – 74 . 38. Zhu GG Stenback F Risteli L et

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in American Journal of Veterinary Research