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sciatic nerve block model in rats, 13 and this prolongation was attributed to blockade of the current ( I h ) produced by hyperpolarization-activated cyclic nucleotide-gated (HCN) channels because the effect of dexmedetomidine was inhibited by the

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in American Journal of Veterinary Research

-opioid receptor agonist U50488H, because it causes activation of the monoaminergic pathways in the CNS, likely increasing concentrations of serotonin and resulting in awareness. Tramadol-induced inhibition of norepinephrine reuptake is similar to inhibition by (α

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in American Journal of Veterinary Research

more intensive pain management for several days’ duration. Untreated or poorly treated pain leads to activation of the sympathetic nervous system, causing elevations in cortisol, norepinephrine, and epinephrine. 1 Prolonged elevation of these “fight

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in American Journal of Veterinary Research