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Hypertrophic cardiomyopathy is the most common form of heart disease in cats. Multiple genetic mutations have been identified in humans with HCM; however, to date, a myosin-binding protein C mutation in Maine Coon cats and Ragdoll cats is the only

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in American Journal of Veterinary Research

compromise diastolic LV function. 1–3 Treatment of cats with subclinical HCM remains controversial but has been directed at relieving obstruction of the LV outflow tract, improving LV diastolic function, and preventing arterial thromboembolism and sudden

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in American Journal of Veterinary Research

significant difference in plasma ANP concentration between cats with HCM and healthy control cats was detected. However, results of 2 other recent studies 36,37 indicated that plasma ANP concentrations were significantly increased in cats with CM, compared

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in American Journal of Veterinary Research